RE: [FH] Moomoo
- --- Shane Fox Luper <sfoxluper@...> wrote:
> Oh Amy, I am heartbroken for you. I am so sorry forThank you all for your wonderful e-mails. It makes me
> your loss of Moomoo. We
> call our cat Maggie "Moomoo" too. :-( I am sure you
> will find some help in
> this group. Many knowledgeable people, including a
> vet are here. I can't say
> that I have a lot of knowledge with what you and
> Moomoo experienced.
> Can you tell us who the first vet/cardiologist is?
> Take good care. You and Moomoo are in my thoughts.
feel better to read them. I'm still trying to
understand fully what happened to Moo -- the more I
know, the better I feel about it. My frame of
reference -- as is all of ours -- is people -- and
obviously heart disease in cats seems very different
than in humans. I understand that it's harder to
diagnose and the same screening tests aren't done as
The first vet who was the one on call the Monday night
we took Moo in with her first episode was a Dr.
Barshovsky (in NYC). She didn't communicate very well
with me and I often felt like she was simply annoyed
by all of my questions. I'm sure she did the best she
could for Moo --- after all, Moo did live through
Monday night, and in retrospect, that seems lucky. The
first cardiologist was a Dr. Kramer --- I never got
the chance to speak to him myself and ask questions --
this might have been part of why I wasn't clear on
what was actually going on. The vet relayed the
treatment to me, but gave me a copy of the first echo
report which actually said to give the medication on a
"trial basis" --- so perhaps this indicated doubt on
his part as well regarding Moo's prognosis.
The second cardiologist who I used was at the Animal
Medical Center -- an absolutely fabulous place. Her
name was Dr. Bond. I loved her. She was honest with
me, but in a kind way. She explained everything very
thoroughly which made me trust that she was doing
everything possible for Moo. I asked her why her
diagnosis/prognosis seemed so different from the first
one, and she said that they weren't so different: the
echo reports were the same, the first cardiologist
just chose a different type of medicine and did not
relay the high possibility of Moomoo dying so soon.
Certainly the first vet did not say that she was at
such high risk for these episodes happening at any
time. But perhaps she couldn't bring herself to say
that? I just think she was a bad communicator.
> From: amy brightfield=== message truncated ===
> Sent: Friday, June 04, 2004 8:06 AM
> To: email@example.com
> Subject: [FH] Moomoo
> Hello everyone:
> My little black and tan tabby, moomoo, just passed
> away this past Sunday from severe heart disease. She
> was just eight years old. I wanted to tell you her
> story in hopes that you all may glean some new
> information about heart disease.
> She was diagnosed with arrhythmic right ventricular
> cardiomyopathy. What basically happened was that she
> had what the cardiologist thinks was a heart attack
> last Monday night. This caused an arrhythmia. She
> never had any problems before this; I took her to a
> yearly check-up and the vet never detected anything
> abnormal. (Although now I tend to think maybe she
> missed something?)
> She survived the h. attack on Monday night. THey did
> bloodwork on her Tues. and an echocardiogram on
> Wednesday which reported right and left ventricle
> dilation; some thickening on the left wall and the
> arrhythmia. That vet sent Moomoo home with diltiazem
> (a calcium channel blocker) and told me she could
> up to five years.
> I wasn't keen on how this vet center had treated
> so I took her to another cardiologist on Thursday
> did another EKG and an echo. She came in and told me
> that Moomoo might only have days to live. I was in
> total shock. Here I had just come from the other vet
> who told me it might be years and that she didn't
> to come back for another echo for six months! But
> new cardiologist was definitely much smarter.
> She explained to me that because Moomoo had so much
> wrong with her heart; the dilation of the
> the thickening of the wall and the arrhythmia ---
> her condition was very hard to treat. Because of the
> arrhythmia, she was at high risk for sudden cardiac
> death and/or throwing a clot at any time. That's why
> her prognosis was so poor.
> But she did send Moomoo home with medicine --
> Enapapril (Sp?) and also instructed me to give her
> mg of aspirin every three days. She said she hoped
> that Moomoo would recover from the heart attack and
> that the medicine would help improve her heart's
> We gave her the aspirin and heart medicine thursday
> night and she ate a very small amount. That was the
> last time she really ate anything. I called the
> cardiologist on Sat morning and she prescribed an
> appetite stimulant, but told me that if Moomoo
> start eating by Sunday am that we should put a
> tube in because cats can develop liver disease if
> don't eat for just two-three days.
> Moomoo was extremely lethargic and not herself at
> she hadn't meowed since the Monday night episode
> she was meowing in pain.
> We ended up putting the feeding tube in sunday am,
> took her home and gave her two feedings. This just
> broke our hearts to see her with the collar she had
> wear to make sure she didn't rip the tube out. But I
> felt like if we were going to give her every chance
> live, we had to try the feeding tube or else another
> fatal problem --- the liver disease -- would
> Sunday night she had an episode of open-mouth
> breathing and we rushed her to the emergency room
> where she went into cardiac arrest.
> I spoke to the cardiologist this week and she said
> thinks that Moomoo's heart just couldn't recover
> the heart attack last Monday as she had hoped it
> and it eventually spiraled into a fatal arrhythmia
> which caused the cardiac arrest.
> From everything I've read, it doesn't seem like
> catching this condition early would have improved
> prognosis much. But she didn't just have the HCM --
> she had all of these other things going on.
> Has anyone else heard of this? Could the thickening
> the walls lead to a heart attack? Is a murmur often
> NOT detected until heart disease has already
> Thank you for reading moomoo's story. We miss her
> dearly. She was a sweet, affectionate cat.
> --- Susan <somnamblst@...> wrote:
> > Great news Morticia,
> > Can you tell us if Spazz is on an ACE-inhibitor?
> > so
> > is he also on lasix?
> > The reason I ask is because there is one
> > cardiologist
> > (Rush) who has reported improvement he has
> > attributed
> > to an ACE-inhibitor.
> > "A recent report by Rush, et al. demonstrated a
> > reduction in wall thickness with the
> > of
> > enalapril to cats with HCM. This suggests a
> > potential
> > role for ACE-inhibitors in the treatment of HCM.
> > These drugs are generally safe and do play a role
> > cases which are refractory or in which pleural
> > effusion is present. In asymptomatic patients,
> > is
> > logical that the renin-angiotensin-aldosterone
> > system
> > is not pathologically activated, and hence
> > ACE-inhibitors would not be useful. The study
> > referred to argues that they may play a role,
> > however.
> > Further studies are being planned."
> > Susan
> > --- Morticia <morticiaw666@...> wrote:
> > > Good Morning All,
> > >
> > >
> > > Just wanted to give you the great news we got
> > > yesterday. Spazz went
> > > in for his 6 month check up, and his heart has
> > > improved :-)
> > > The chamber on the left that was holding the
> > > in is now pumping
> > > the blood out more like it supposed to. Dr.
> > > did tell me that
> > > he is still a very sick little boy, but that he
> > > very optimistic
> > > on his condition as it is now. The heart walls
> > that
> > > had started to
> > > thicken has now thinned out slightly. What
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